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1 According to this theory, a chronic, low-level deficiency of a nutrient like EGT might not cause an acute, overt disease but may lead to cellular damage that accelerates the onset of age-related chronic illnesses like cardiovascular diseases (CVDs), cancers, and metabolic syndromes

Thus, the use of combined medications to address the drug resistance arising from Keap1 mutations represents a promising novel approach to combat chemotherapy resistance in clinical settings

We postulate that NAD + /NADH disequilibrium in the gdh3 / gdh3 mutant led to a different proline catabolism in the mutant compared to the wild-type strain